Literaturverzeichnis

1. Alberts, B., Bray, D., Lewis, J., Raff, M., Roberts, K., Watson, J.D. (1990/95): Molecular biology of the cell. 2. Aufl. Weinheim, Basel, Schweiz.
2. Agapitos, E., Kavautzas, N., Nauas, J., Margiri, Z., Bakouris, M., Kassis, K., Panolaridis, A., Davaris, P. (1996): The myocardial fibrosis in patients with dilated cardiomyopathy. The application of image analysis in the myocardial biopsies. Gen.- Diagn.- Pathol. 141 (5 - 6), 305 - 11.
3. Bajusz, E., Hamburger, F., Baker, J.B., Opie, L.H. (1966): The heart muscle in muscular dystrophy with special reference to involvement of the cardiovascular system in the hereditary myopathy of the hamster. Ann. N.Y. Acad. Sci., New York, 138, 213 - 231.
4. Bajusz, E. und Lossnitzer, K. (1968): Ein neues Krankheitsmodell: Erbliche nicht vasculare Myocarddegeneration mit Herzinsuffizienz. Münch. Med.Wochenschr., München, 31, 1756 - 1768.
5. Bergmann, G.P. (1990): Fibronectin und Fibronectinbindungsaktivitäten von pferdepathogenen Streptokokken der Serogruppe C und derene mögliche Bedeutung bei Phagozytose und Adhärenz. Medizinische Dissertation, Veterinärmedizin, J. - Liebig - Universität Gießen.
6. Blumenstock, F.A., Saba, T.M., Weber, P., Laffin, R. (1978): Biochemical and immunological characterisation of human opsonic alpha - 2 - SB glycoproteinen: Its identy with weas - onsoluble globin. J. Biol. Chem. 253, 4287 - 4291.
7. Boluyt, M.O., Bing, O.H., Lakatta, E.G. (1995): The ageing spontaneously hypertensive rat as a model of the transition from stable compensatet hypertrophie to heart failure. Eur. - heart -j., 16 Suppl. N., 19 - 30.
8. Britgen, ?., (1957): Uncommon myocardial diseases, the non-coronary cardiomyoapthies. Lancet, London, II, 1179 - 1243.
9. Buck, C.A. und Horowitz, A.F. (1987): Cell surface receptors for extrazellular matrix mulekules. Ann. Rev. Cell. Biol. 3, 179 -205.
10. Büchner, F. und Onishi, S. (1967): Frühstadien der akuten hypoxischen Veränderungen des Herzmuskels im elektronenmikroskopischen Bild und ihre Bedeutung für die akute hypoxische Herzinsuffizienz. Path. Ana. 135, 153 - 182.
11. Bundgaard, M. (1979): Mitochondrienfunktion und Adeninnukleotidgehalt des Herzmuskels in Abhängigkeit von der Ischämiedauer. Medizinische Dissertation an der Fr.-Alexander-Universität. Erlangen - Nürnberg. 37 - 56.
12. Bundgaard, M. (1980): Transport pathway in Capillaries - in search of pores. Ann. Rev. Physiol., Stanford, 42, 325 - 336.
13. Chang, Z.L., Beezhold, D.H., Personin, C., Snen, Z.L. (1992): Fibronectin cell binding domain triggered transmembrane signal transduction in human monocytes. J. Leucocyte Biol. 53, 79 - 85.
14. Collip, P.J. und Chen, S.Y. (1981): Cardiomyopathy and selenium deficiency in a two - year alt girls. New Engl. J. Med., Boston, 304, 1304 - 1309.
15. Cottier, H. (1992): Pathogenese, Bd. 1. Springer -Verlag, 101 - 104.
16. Davis, L.S., Oppenheimer - Marks, N., Badarczyk, J.L., Mclutyre, B.W., Lipsky, P.E. (1990): Fibronectin promotes proliferation of naive and memory T - cell by signaling thourg the VLA - and VLA - 5 integrins molekules. J. Imm. 145, 785 - 793.
17. Döring, V., Baumgarten, H.G., Pokar, H., Gercken, G. (1975): Metabolism and fine structure of the magnesium - procaine arrested perfused heart. Basic Res. Cardiol. 70, 186 - 197.
18. Faber, J.L. (1983): The role of calcium in cell death. Life Sci., NewYork, 29, 1289 - 1295.
19. Factor, S.M., Minase, T., Cho, S., Dominitz, R., Sonnenblick, E.H. (1982): Microvascular spasm in the cardiomyopathic syrian hamster: A preventable cause of focal myocardial necrosis. Circul., NewYork, 66, 342 - 354.
20. Feder, E. (1976): Die Auswirkung von sechszig - Minuten - dauernder hypothermer Ischämie auf das isolierte, isovolumetrisch arbeitende Hundeherz. Med. Dissertation, L.- Maximilian - Universität München.
21. Ferrans, V.J., Buja, L.M., Maron, B.J. (1975): Myofibrillar abnormalities folleowing cardiac muscle cell injury. In Fleckstein, A. und Grona, G. (eds.): Recent advances in studies on cardiac structure and metabolism. Vol. VI. Urban und Schwarzenberg. München - Berlin - Wien, 367 - 382.
22. Ferrans, V.J., Massuri, R.A., Shugoll, G.I., Ali, N., Roberts, W.C. (1973): Ultrastuctural studies of myocardial biopsies in 45 patients with obstructive or congestive cardiomyopathy In: Bajusz, E. und Rona, G. (eds.): Recent et advances in studies on cardiac structure and metabolism. Vol. II University Park Presp., Baltimore, 231 - 271.
23. Figulla, H.R., Vetterlein, F., Glaubitz, M. Krenzer, H. (1987): Inhomogenous capillary flow and its prevention by Verapamil and Hydralacine in cardiomyopatic syrian hamster. Circ. NewYork, 76, 208 - 216.
24. Finkel, M.S., Shen, L., Oddis, C.V., Romeo, R.C. (1993): Verapamil regulation of a defective SR release channel in the cardiomyopathic syrian hamster. Life Sci., NewYork, 52, 1109 -1119.
25. Fleckstein, A. (1971): Specific inhibitors ant promotors of calcium action in the excitation - contraction coupling of heart muscle and their role in the prevention or production of myocardial lesions. In: Harris, P. und Opie, L. (ets.): Calcium and the heart. Acad. Press. London - NewYork, 135 - 188.
26. Fleckstein, A., Janke, J., Döring, H.J., Leder, O. (1974): Myocardial fiber necrosis due to intracellular calcium overload - a new principle in cardiac pathophysiology. In: Dhalla, N.S. (eds.): Recent advances in studies on cardiac structure and metabolism. Vol. IV. University Park Press, Baltimore - London - Tokio, 563 - 580.
27. Fleckstein, A., Janke, J., Döring, H.J., Pachinger, O. (1973): Calcium over load as the determinant factor in the production of catecholamin - induced myocardial lesions. In: Bajusz, E. und Rona, G. (eds.): Recent advaces in studies on cardiac structure and metabolism. Vol. II University Park Press, Baltimore, 455 -466.
28. Fleckstein, A.. und Rona, G. (1975): Recent advances in studies on cardiac structure on metabolism. Vol. VI, Pathophysiology and morphology of myocardial all alterations. München - Wien - Berlin, Urban und Schwarzenberger.
29. Forman, R. Parmley, W.W., Sonnenblick, E.H. (1972): Myocardial contractility in relation to hypertrophy and failure in myopathic syrian hamster. J. Molec. Cell. Cardiol., London - NewYork, 4, 203 - 211.
30. Frenzel, H., Kasper, M., Kuhn, H., Losse, B., Reifschneider, G., Hort, W. (1985): Licht- und elektronenmikroskopische Befunde in Früh- und Spätstadien der Herzinsuffizienz. Untersuchungen an Endomyocardbiopsien von Patienten mit latenter und dillatativer Cardiomyopathie. Zeitschr. Kardiol., Darmstadt, 74, 135 -143.
31. Fuchs, U. (1964): Elektronenmikroskopische Untersuchungen an Blutkapillaren. Med. Habil.-Schrift. Leipzig.
32. Gabler, U., Berndt, A., Kosmehl, H., Mandel, U., Zardi, L., Muller, S., Stelzner, A., Katenkamp, P. (1996): Matrix remodelling in delated cardiomyopathy entails the occurrence of oncofetal fibronectin molecular variants. Heart. , 75, 358 - 362.
33. Galambos, J.T. (1977): The rate of synthesis of glycosaminoglycans and collagen by fibroblasts cuttured from adult human liver biopsies. J. Clin. Invest. ,60, 107.
34. Goodwin, J.F. (1974): Prospects and predection for the cardiomyopathy in circulation. J.Molec. Cell. Cardiol., NewYork, 50, 210 - 219.
35. Goodwin, J.F. (1982): The frontiers of cardiomyopathy. Brit Heart J.., London, 48, 1 - 18
36. Goodwin, J.F. (1985): Mechanisms in cardiomyopathies. J. Molec. Cell. Cardiol., London - NewYork, 17, Suppl. 2, 5 - 9.
37. Goodwin, J.F. und Oakley, C.M. (1972): The cardiomypathies. Brit. Heart J., London, 34, 545 - 552.
38. Hauschild, U., Baghirzade, M. F., Kirsch, U. (1970): Kapillarkompression al Ischämiefolge. Virchow.- Arch. Abt. A. Path. Anat., 351, 205 - 224.
39. Hecht, A. (1980): Spezielle Pathologie. Verlag Volk und Gesundheit, 60 - 61.
40. Hörmann, H. (1985): Fibronectin and phagocytosis. Blut, 52, 307 - 314.
41. Hort, W. (1955): Quantitative Untersuchungen über die Kapillarisierung des Muskels im Erwachsenen- und Greisenalter bei Hypertrophie und Hyperplasie. Virchow - Arch. Abt. A. Path. Anat., 327, 560.
42. Hort, W. und Severidt, H. J. (1966): Kapillarisierung und mikroskopische Veränderungen im Myocard angebohrener Herzfehler. Virchow - Arch. Abt. A. Path. Anat., 341, 192 - 204.
43. Hynes, R.O. (1987): Integrins. A family of cell syrface receptors. Cell. , 48, 549 - 554.
44. Jasmin, G. und Bajusz, E. (1975): Prävention of myocardial degeneration in hamsters with hereditary cardiomyopathy. In: Fleckstein, A. und Rona, G. (eds.): Recent advances in studies on cardiac structure an metabolism. München, Vol. VI. Urban und Schwarzenberg, 219 - 229.
45. Jasmin, G. und Eu, H. Y. (1979): Cardiomyopathie of hamsters dystrophy. Ann. NewYork Acad. Sci., NewYork, 317, 46 - 58.
46. Jasmin, G. und Proschek, L. (1980): Prevention of myocardial degeneration in hamsters with hereditäry cardiomyopathy. In: Fleckstein, A. und Roskamm, H. (eds.): Calcium antagonismus. Berlin - Heidelberg - NewYork, Springer - Verlag, 144 - 150.
47. Jasmin, G. und Proschek, L. (1982): Hereditary polymyopathy and cardiomyopathy in the syrian hamster. I. Progression of heart an skeletal muscle lesions in the UM - X 7.1. line. Muscle and nerve, Boston, 5, 20 - 25.
48. Jasmin, G. und Proschek, L. (1984): Calcium and myocardial cell injury. An appraisal in the cardiomyopathic hamster. Can. J. Physiol. Pharmacol., Ottawa, 62, 891 - 898.
49. Kewley, M. A. (1978): Studies of elasic tissue formation in the developing bovine ligamentum nucheae. J. Pathol. ,124, 95.
50. Khuchua, Z. A., Vasiljeva, E. V., Clark, J. F., Kovchazkhina, O. V., Branishte, T., Kapelko, V. I., Kuznetsov, A. V. (1992): The creatine kinase system and cardiomyopathy. Ann. J. Cardiovasc. Pathol., NewYork , 4, 223 - 234.
51. Knieriem, H., J. (1978): Electron mikroscopic findings in congestive cardiomyopathie. In: Kaltenbach, M., Loogen, F., Olsen, E., G., J. (eds.): Cardiomyopathy and myocardial biopsy. Berlin - Heidelberg - NewYork, Springer - Verlag, 71 - 86.
52. Kramer, R. H., McDonald, K. A., Vu, M. P. (1989): Human melanoma cell express a novel integrin receptor for laminin. J. Biol. Chem., 264, 15642 - 15649.
53. Kunkel, B., Kober, G., Lapp, H., Leuschner, U., Kaltenbach, M. (1977): Klinische, licht- und elektronenmikroskopische Befunde bei frühen und fortgeschrittenen Formen kongestiver Cardiomyopathie. Z. Kardiol. Darmstadt, 66, 198 - 202.
54. Kunkel, B., Schneider, M., Eisenmenger, M., Bergmann, A., Hopf, B., Kaltenbach, M. (1987): Myocardial biopsy in patients with hypertrophic cardiomyopathy: Correlations between morphologic and clinical parameters and development of myocardial hypertrophy under medical therapy. Z. Kardiol. ,76 Suppl. 3, 33 - 38.
55. Kunkel, B., Schneider, M., Hopf, R., Hübner, K. (1985): Wiederholte Myocardbiopsien bei Patienten mit hypertrophischer Cardiomyopathie und medikamentöser Therapie.
56. Verh. Dt. Ges. Pathol. Stuttgart, 69, 423 - 427.
57. Kunkel, B., Schneider, M., Kober, G., Bussman, W.D., Hopf, R., Kaltenbach, M. (1982): Die Morphologie der Myocardbiopsie und ihre klinische Bedeutung. Zeitschr. Kardiol., Darmstadt, 71, 787 - 794.
58. Landes, F.M. und Pappenheimer, J. R. (1963): Exchangs of substances through the capillary walls. In: Handbook of Physiology. Fds: Hamilton, W. F. und Low, P., Sect. 2, Circulation 2, Baltimore: Williams and Wilkins, 961 - 1034.
59. Layman, D. L. und Titus, J. L. (1975): Synthesis of typ I collagen by human smooth muscle cells in vitro. Lab. Invest., 33, 103.
60. Lindenmayer, G. E., Harigaya, S., Bajusz, E., Schwartz, A. (1970): Oxydative phosphorylation and calcium transport of mitochondria isolated from cardiomyopathic hamster and heart. J. Mol. Cell. Cardiol., London - NewYork, 1, 249 - 259.
61. Linzbach, A. J. (1947): Mikrometrische und histologische Analyse hypertrophierter menschlicher Herzen. Virchow - Arch. Path. Anat., Berlin - West, 314, 534 - 595.
62. Lochner, A., Brink, A. J., van der Walt, J. J. (1970): The significance of biochemical and structural changes in the development of the cardiomyopathie of the syrian hamster. J. Mol. Cell. Cardiol., London - NewYork, 1, 47 - 64.
63. Löffler, G. und Petrides, P.E. (1997): Biochemie und Pathobiochemie, Springer-Verlag, 736 - 738.
64. Lossnitzer, K., Janke, J., Hein, B., Strauch, H., Fleckstein, A. (1975): Disturbed myocardial calcium metabolism: A possible pathogenetic factor in the hereditary cardiomyopathy of the syrian hamster. In: Fleckstein, A. und Rona, G. (eds.): Recent advances instudies on cardiac structure and metabolism. München, Voll. VI. Urban und Schwarzenberg, 207 - 217.
65. Lunkenheimer, P. P. und Merker, H. J. (1973): Morphologische Studien zur funktionellen Anatomie der "Sinusoide" im Myocard. Z. Ana. Entw.-Gesch., 142, 65 - 90.
66. Majno, G. und Pallade, G. E. (1961): Studies of inflammation. The effect of histamine and serotonin of vascular permeabily: A electron microscopic study. J. biophys. and biochem. Cytol., Baltimore, 11, 571 - 606.
67. Matsumori, A. und Akiwa (1996): Cytocines in myocarditis and cardiomyopathies. Z. Cardiol., 11, 302 - 309.
68. Matsumori, A. und Sasayama, S. (1996): Role of cytokines in the syndrome of heart failure. Intern. Med., 35 (1), 60 - 63.
69. Matturi, L., Biondo, B., Grosso, E., Lavezzi, A. M., Rossi, L. (1995): Morphometric and densitometric approach in hypertrophic cardiomyopathy. Eur. J. Histochem., 39, 237 - 244.
70. McCully, J. D., Mably, J. D., Sole, M. J., Liew, C. C. (1991). RNA transcription and translation in the heart of normal and cardiomyopathic syrian hamsters. Biochem. Cell. Biol., Ottawa, 69, 88 - 91.
71. McDouglas, J. S. und Azar, H. A. (1972): Tritiated proline in macrophages. Arch. Pathol., 93, 13.
72. Mohr, W. und Lossnitzer, K. (1972): Morphologische und klinische Manifestation der hereditären Myopathie beim syrischen Goldhamster ( BIO 8262). Verl. Dt. Ges. Pathol., Stuttgart, 56, 645 - 653.
73. Mosher, D. F. (1984): Physiology of fibronectin. Ann. Rev. Med. , 35, 561 - 575.
74. Mosseri, M., Schaper, J., Admon, D., Hasin, Y., Gotsman, M. S., Sapoznikov, D., Pickering, J. G., Yarom, R. (1991): Coronary capillary in patiens with congestiver Cardiomyopathy or Angina pectoris with patent main coronary arteries. Z. Circ. Vol. 84, 1, 203 - 210.
75. Nayler, W. G. (1981): The circle of cacium in the ischamic myocardium. Am. J. Pathol., NewYork, 102, 262 - 270.
76. Okada, H., Kawaguchi, H., Kaudo, T., Sawa, H., Okamoto, H., Watanabe, S., Ursawa, K., Murakami, T., Kitabataka, A., (1996): Alteration of extrazellular matrix in dilated cardiomyopathic hamster heart. Mol. Cell. Biochem., 156, 9 - 15.
77. Olsen, E. G. J. (1973): The pathology of the heart. Stuttgart, Thieme - Verlag.
78. Olsen, E. G. J. (1980): Morphologic evaluation (histological, histochemical, ultrastructural) of endomyocardial biopsies. In: Bolte, H. D. und Dietrich, H. (eds.): Myocardial biopsies Berlin, Springer - Verlag, 13 -19.
79. Palade, G. E. (1961): Blood capllaries of the heart and other organs. Circ. 24, Philadelphia, 368 - 384.
80. Pappenheimer, J. R. (1953): Passags of molecules through capillary walls. Physiol. Rev., Baltimore, 33, 387 - 423.
81. Paterson, R. A., Layberry, R. A., Nadkarni, B. B. (1972): Cardiac failure in the hamster. A biochemical and electron microscopical study. Lab. Invest., NewYork, 26, 755 - 766.
82. Pearse, A. C. E. (1964): The histochemistry and electron microscopy of obstructive cardiomyopathy. In: Wolstenholme, G. E. und O´Connor, M. O. (eds.): Cardiomyopathies. CIBA Found. Symp., J. & A. Churchill. London, 132 139.
83. Perloff, J. K. (1981): Pathogenesis of hypertrophic cardiomyopathy. Hypotheses and speculation. Am. Heart.- J., St. Louis, 101, 219 -226.
84. Pomerance, A. und Davis, M.J. (1975): The Pathology of the heart. Scienc. Publ., Blackwell, 7.
85. Proctor, R. A. (1987): Fibronectin: A brief overview of its structure function and physiology. Rev. Infect. Dis., 9, 317 - 321.
86. Pscherembel (1992): Klinisches Wörterbuch, Gruyter - Verlag, Berlin - NewYork 256. Aufl.
87. Pytela, R., Pierschbacher, M.-D., Ruoslahti, E. (1985): Identification and isolation of a 140 kd cell surface glycoprotein with properties expected of a fibronectin receptor. Cell., 40, 191 - 198.
88. Reichenbach, D.D. und Benditt, E.P. (1970): Catecholamines and cardiomyopathy: The pathogenesis and potential importance of myofibrillar degeneration. Human Pathol., Philadelphia, 1, 125 - 150.
89. Roberts,W.C. und Ferrans, V.J. (1975): Pathologic anatomy of the cardiomyopathies. Ideopathic dilated and hypertrophic types, infiltrative types and endomyocardial disease with and without eosinuphilia. Human Pathol., Philadelphia, 278 - 302.
90. Rösen, P., Ballhausen, T., Block,W., Addicks, K. (1995): Endothelial relxation is disturbed by oxidative stress in the diabetic heart: influence of tocopherol as antioxidant. Diabet., 38 (10), 1157 - 1168.
91. Ruoslahti, E. (1991): Integrins. J. Clin. Invest., 95, 1 - 5.
92. Sabbah, H.N., Sharov, V.G., Lesch, M., Goldstein, S. (1995): Proggression of heart failure: a role for interstitial fibrosis. Mol. Cell. Biochem., 147, 29 - 34.
93. Sakanashi, T., Sato, S., Nozuhara, A., Adachi, K., Okamoto, T. Koga, Y., Toshima, H. (1991): Vitamin E deficiency has a pathological role in myocytolysis in cardiomyopathic syrian hamster (BIO 14.6.). Biochem. Biophys. Res. Comm. , NewYork, 181, 145 -150.
94. Sapp, J.L. und Howlett, S.E. (1994): Density of ryanodine receptors is increased in Sarcoplasmatic reticulum from prehypertrophic cardiomyopathic hamster heart. J. Mol. Cel. Cardiol., London - NewYork, 26, 325 - 334.
95. Sasayama, S. (1991): Recent progress in failing heart syndrome. H. Sugar (eds.), Springer - Verlag Tokio, Berlin, IX.
96. Schaper, J., Mollnau, H., Hein, S., Scholz, D., Munkel, B., Deveaux, B. (1995): Wechselwirkungen zwischen Cardiomyozyten und der extrazellulären Matrix im insuffizienten menschlichen Herzen. Z. Kardiol., 84, Suppl. 4, 33 - 38.
97. Schaper, J. und Speiser,B. (1990): The extracellular matrix in the failing human heart. Basic - Res. - Cardiol., 87 (1), 303 - 309.
98. Schaper, J., Speiser, B., Brand, T. (1993): The Cytoskeleton and extracellular matrix in Human Heart with Dilated Cardiomyopathy. In: Figulla et al. (eds.): Ideopathic Dilated Cardiomyopathy. Springer - Verlag, Berlin - Heidelberg.
99. Schehrag, A. (1995): Neues aus der Herz- Kreislaufforschung. Therapie - Woche 18; 1030 - 1031.
100. Schölmerich, P., Just, H., Meinertz, T. (1996): Handbuch der Medizin: Myocarderkrankung, Pericarderkrankung, Herztumoren. Springer - Verlag.
101. Schoenmachers, J. (1949): Die Herzschlagadern bei der arteriokardialen Hypertrophie. Z. Kreisl. Forsch., 38, 321 - 336.
102. Schwartz, A., Sordahl, L.A., Crow, C.A., McCollum, W.B., Harigaya, C., Bajusz, E. (1972): Several biochemical characteristics of the cardiomyopathic syrian hamster. In: Bajusz, E. und Rona, G. (eds.): Recent advances in studies on cardiac strukture and metabolism. Vol. I. Urban & Schwarzenberg, Baltimore, 235 - 242.
103. Simionescu, M., Simionecu, N., Palade, G.E. (1974): Structural basis of permeability in sequental segmente of the Microvasculature. II Pathways followed by microperoxydase acroses the endothelium. Microvascular Research , NewYork, 15, 17 - 36.
104. Sole, M.J. und Liew, C.C. (1988): Catecholamines, calcium and cardiomyopathy. Am. J. Cardiol., NewYork, 62, 20 - 24.
105. Sonnenblick, E.H., Fein, F., Capasso, J.M., Factor, S.M. (1985): Microvascular spasm as a cause of cardiomyopathies and the calcium blocking agent Verapamil as potential promary
106. therapy. Am. J. Cardiol., 55, 179 - 184.
107. Speiser, B., Riess, C.F., Schaper, J. (1991): The extracellular matrix in Human Myocardium. Part. I. Collagen I, III, IV, VI. Cardiosience Vol 2, 4, 225 - 232.
108. Spinale, F.G., Burchell, S.A., Crawford, F.A., Tanaka, R., Zile, M.R. (1992): Effect of chronic tachycardia - induced cardiomyopathy on the beta - adrenergic receptor System. Z. - Thorac. - Cardiovasc. - Surg., 104 (4), 1006 - 1012.
109. Spinale, F.G:, Grine, R.C., Tempel, G.E., Crawford, F.A., Zile, M.R. (1992): Alterations in the myocardial capillary vasculature accompany tachycardia - induced Cardiomyopathy. Basic - Res - Cardiol., 87 (1), 65 - 79.
110. Steahelin, A.L. (1974): Structure and function of intracellular junctions. Internat. Rev. Cytol., NewYork, 39, 191 - 283.
111. Strauch, M. und Lossnitzer, K. (1975): Left ventricular function in syrian hamsters of diffrent ages with hereditary cardiomyopathy. In: Fleckstein, A. und Rona, G. (eds.): Recent advances in studies on cardiac strukturs and metabolism. Vol. VI Urban & Schwarzenberg, München - Berlin - Wien, 283 - 290.
112. Tawada - Iwata, Y.T., Imagawa, T., Yoshida, A., Takahashi, M., Nakamura, H., Shigekawa, M. (1993): Mechanicel extractions of T - tubule/junctional SR from cardiomyopathic hamster heart. Am. J. Physiol., New York, 264, 1447 - 1453.
113. Trevi, G., Sheibam, J., Gorni, A. (1995): Myocardial hypertrophy and arterial hypertention. G. Ital. Cardiol., 25 (10), 1331 - 1338.
114. Vogelberg, K. (1957): Die Lichtungsweite der Coronarostien an normalen und hypertrophen Herzen. Z. Kreislaufforsch., 46, 101.
115. Wada, A., Yoneda, H., Shibata, N., Iuni, Y., Onishi, S. (1975): Morphological and histochemical studies on the heart of cardiomyopathic syrian hamster. In: Fleckstein, A. und Rona, G. (eds.): Recent advances studies on cardiac structure and metabolism. Vol. VI, Urban & Schwarzenberg, München - Berlin - Wien, 275 - 282.
116. Wagner, H.J., Sax, F.L., Weißmann, H.F., Porterfield, J., McIntosh, C., Weißfeldt, M.L., Snyder, S.H., Epstein, S.E. (1989): Cacium - antagonist receptors in the atrial tissue of patients with hypertrophic cardiomyopathy. N. Engl. - J. Med., 320 (12), 755 - 761.
117. Wearn, J.T. (1928): Quantitative Untersuchungen über die Kapillarisierung des Muskels im Erwachsenen- und Greisenalter bei Hypertrophie und Hyperplasie. J. of Exper. Med., 47, 273.
118. Weihrauch, D., Arras, M., Zimmermann, R., Schaper, J. (1995): Importance of myozytes/makrophages and fibroblasts for healing of micronecrosis in porcine myocardium. Mol. and Cell. Biochem., 147, 13 - 19.
119. Weisman, H.F. (1993): The role of calcium channel abnormalities in syrian hamster cardiomyopathy. Clin. Imm., Imm. Path., 68, 170 - 174.
120. Welt, K. (1984): Zur Permeabilität von Myocard -und Skelettmuskelkapillaren der weißen Ratten verschiedener Ontogenese - Stadien für wasserlösliche Makromoleküle. Eine elektronenmikroskopische Studie mit Tracer- und Gefrierätztechnik. Habil. - Schrift, Anat. Inst. Univ. Lpz.
121. Wissing, S.L. (1981): Functions of endothelial junctions. Adv. Physiol. Sci. Vol., 7 Pergamon Pres. Acadmai Kiado, Budapest, 133 - 144.
122. Wrogeman, K. und Nyler, E.G. (1978): Mitochondrial calcium overloading in cardiomyopathic hamsters. Mol. Cell. Cardiol., London, 10, 185 -195.
123. Wrogeman, K. und Pena, S.P.J. (1976): Mitochondrial calcium overload: A general mechanism for cell necrosis in muscle diseasis. Lancet, London, 1, 672 - 677.
124. York, C.M., Cantrett, C. R., Borum, P.R. (1983): Cardiac carnitine deficienties and alterated carnitine transport in cardiomyopathic hamsters. Arch. Biochem. Biophys., NewYork, 221, 526 - 533.
125. Zebe, M., Schwartz, F., Mall, G., Blickle, J., Dürr, C., Kübler, W. (1984): Die Bedeutung morphologischer Befunde bei der kongestiven Cardiomyopathie. Z. Kardiol., Darmstadt, 73, 181 - 187.
126. Zimmermann, K.U. (1923): Der feine Bau der Blutkapillaren. Z. Anat. und Entw.-Gesch., 68.